Timely - Dr. Mercola offers foods from the list that can be non-organic and have low levels of pesticides:
[
articles.mercola.com]
Be sure to read what he reports about nutrient value etc.
Just as we see controversy over the benefits of supplemental vitamins, minerals and other nutrients in functional medicine versus pharmaceutical medicine, the controversy over nutrient value of organic v. conventionally grown food will continue to rage on because of the very obvious problems involved with how the truth impacts the bottom line. Once again, awareness becomes a key issue and then we all need to make informed choices when options are available. We do the best we can under our own circumstances.
On the toxic chemical burden, however, as pointed out in the very excellent article (following) on environmental influences on health and discoveries in the new field of Epigenetics, it is important also, to be aware that environmental toxins such as pesticides on foods or toxic fluoride in the water have a far-reaching impact on the health of everyone including generations forward. Its not just about eating a couple apples laced with pesticides. Its about a lifetime accumulation and that impact that may even be with us before we were born.
Neurosurgeon, Russell L. Blaylock, MD, has devoted his life to creating awareness over the neurotoxic effects from excitotoxins based on his observations during attempts to prolong the lives of those affected by such exposure. He reminds us that it is not typically just one toxin that causes the problem, but the fact that we are bombarded with multiple doses of multiple substances and combinations have the potential to do significant harm. Its not only infants who are placed at risk for toxic burden.
The liver can only detoxify so much. Toxins have an affinity for fatty tissue like brain, liver and adipose accumulations so we must consider the impact of a lifetime of accumulation. Even worse, infants today are bombarded with vaccines that contain harmful residues that their small organ systems cannot always process efficiently. Its not just infants.
A considerable amount of research indicates that Gulf War Syndrome (GWS), as well as autism, is triggered by combining too many vaccines over too short a period of time. This error is compounded by numerous other toxic events, especially in the Gulf War veteran. These events include exposure to pesticides, aspartame breakdown products, combat stress, high intake of food-based excitotoxins, possible exposure to released nerve agents, and exposure to contaminated vaccines. (Blaylock)
Throughout the entire period since the first Gulf War the Pentagon has been reluctant to admit to a connection between this devastating syndrome which has left tens of thousands of soldiers and their families chronically ill and many of their children deformed and military policies on vaccination. Our soldiers were given approximately 17 vaccinations during a short period of time, despite manufacturers warnings that many of the vaccines were to be spaced over a one-year period.
Several hypotheses for the cause of this syndrome have been proposed, including neurotoxic and immunotoxic effects of pesticides, aspartame degradation products, released chemical warfare agents, toxins from spent uranium shells, combat stress and vaccines.(Blaylock)
This, alone, sets up populations for the impact of cumulative and additive effects of chemicals that the body does not require for health and we must consider the long-term impact of the whole picture. We need to be aware to avoid what we can if at all possible because of long-term consequences.
In one of his Wellness Reports, he says this:
How Scientists Really Think
It is characteristic of modern science, and especially
medicine, to always look for one central cause
of a problem rather than explore additive effects
or even synergic toxicity of many agents (two toxins
acting on the body together may be much more
dangerous when they work in combination).
Yet medicine is gaining new understanding about
the science of toxin synergy in your body and
finding some surprising effects when as few as two
weak toxins are in your body simultaneously.
For example, it is known that when two weakly
toxic pesticides are used alone, neither causes
Parkinsons syndrome in experimental animals, but
when combined, they can cause full-blown
Parkinsonism very rapidly.
The same is true of fluoride. We know that both
fluoride and aluminum, individually, are brain toxins,
but when combined, as we see in fluoridated
water, the mix constitutes an extremely powerful
brain toxin, destroying numerous neurons in the
part of the brain associated with memory and emotional
functions.
It is rare for government agencies to test potentially
toxic chemicals or even food additives in combination,
despite extensive scientific studies showing
toxic synergy for many such compounds.
As in the examples above, we are seeing more
instances of chemicals causing devastating injury
when combined, yet being either mildly toxic or
even nontoxic when used alone.
Few laymen realize that vaccines contain many
chemical additives in addition to the infectious
organism being targeted. A typical vaccine could
include aluminum, mercury, hydrolyzed proteins,
monosodium glutamate, oils, and many complex
molecules known as immune adjuvants. Several of
these (aluminum, mercury, hydrolyzed protein and
MSG) are known to be directly toxic to the brain.
(Blaylock Wellness Report May 2004)
www.newsmax.com $48/year
RACHEL'S ENVIRONMENT & HEALTH NEWS #819
[
www.rachel.org]
June 9, 2005
A NEW WAY TO INHERIT ENVIRONMENTAL HARM
by Tim Montague*
New research shows that the environment is more important to
health than anyone had imagined. Recent information indicates
that toxic effects on health can be inherited by children and
grandchildren, even when there are no genetic mutations
involved.[1] These inherited changes are caused by subtle
chemical influences, and this new field of scientific inquiry
is called "epigenetics."[2]
Since the 1940s, scientists have known that genes carry
information from one generation to the next, and that genes
gone haywire can cause cancer, diabetes, and other diseases.
But scientists have also known that genes aren't the whole
story because identical twins -- whose genes are identical --
can have very different medical histories. One identical twin
can be perfectly healthy while the other develops schizophrenia
or cancer -- so the environment must play a significant role,
not merely genes.
What's surprising is that scientists are now revealing that
these environmental effects can be passed from one generation
to the next by a process called "epigenetics," with
far-reaching implications for human health. Epigenetics is
showing that environmental influences can be inherited -- even
without any mutations in the genes themselves[1] -- and may
continue to influence the onset of diseases like diabetes,
obesity, mental illness and heart disease, from generation to
generation.
In other words, the cancer you get today may have been caused
by your grandmother's exposure to an industrial poison 50 years
ago, even though your grandmother's genes were not changed by
the exposure.[1] Or the mercury you're eating today in fish may
not harm you directly, but may harm your grandchildren.
This emerging field of epigenetics is causing a revolution in
the understanding of environmental influences on health. The
field is only about 20 years old, but is becoming
well-established. In 2004, the National Institutes of Health
granted $5 million to the Johns Hopkins Medical School in
Baltimore to start the Center for Epigenetics of Common Human
Disease.
The latest information appears in a new study by Michael
Skinner and colleagues at the University of Washington,
published in the June 3 issue of Science magazine. Skinner
found that mother rats exposed to hormone-mimicking chemicals
during pregnancy gave birth to four successive generations of
male offspring with significantly reduced fertility.[3] Only
the first generation of mothers was exposed to a toxin, yet
four generations later the toxic effect could still be
detected.
Prior to this study, scientists had only been able to document
epigenetic effects on the first generation of offspring. These
new findings suggest that harm from toxins in the environment
can be much longer lasting and pervasive than previously known
because they can impact several generations.
And therefore a precautionary approach to toxics is even more
important that previously believed. (See Rachel's 765, 770, 775,
781, 787, 789, 790, 791, 802, 803, 804.)
Over the past sixty years doctors and scientists have pieced
together a picture of the genetic basis for life and some of
the genetic causes of! human and animal disease. Genes regulate
the production of proteins -- the essential building blocks of
life. Genes are composed of a finite series of letters (a code
made up of Cs, Ts, As, and Gs, each representing a nucleotide)
embedded in long strands of DNA. DNA is the large molecule,
composed of genes, that carries the genetic inheritance forward
into the next generation.
There are approximately three billion 'letters' in the human
genetic code. Science has long understood that when a gene
mutates -- that is, when a typo is introduced -- it can have
far-reaching effects for the cell, the tissue and the organism
as a whole. For example, a genetic mutation caused by too much
sun (ultraviolet radiation), could result in abnormal
uncontrolled cell growth which could lead to skin cancer which
could spread throughout your body. Stay in the shade and you
reduce your risk.
But now scientists are seeing that disease can be passed from
generation to generation without any genetic mutations.[1] The
DNA molecule itself gets another molecule attached to it, which
changes the behavior of the genes without changing the genes
themselves.[1] The attachment of these additional molecules is
caused by environmental influences -- but these influences can
then be passed from one generation to the next, if they affect
the germ cells, i.e., the sperm or the egg.
Scientists have, so far, discovered three different kinds of
"epigenetic" changes that can affect the DNA molecule and thus
cause inheritable changes. One is the methyl molecule.
Scientists began to see direct connections between human
diseases like cancer and these subtle genetic variations like
methylation in 1983 when Andrew Feinberg and his colleagues at
Johns Hopkins found that cancer cells had unusually low
incidence of DNA-methylation.[4]
Methyl is a molecule of one carbon atom and three hydrogen
atoms. Together they attach to a strand of DNA altering its
three-dimensional structure and the behavior of specific genes
in the DNA strand. It turns out that methylation works like a
volume control for the activity of individual genes. Whereas
genetic mutations are typos and relatively easy to test for,
epigenetic changes are analogous to the formatting of the text
(e.g. font, size, and color) and are much less-well understood.
Over the past 20 years, Feinberg and many other cancer
specialists have documented the wide-spread influence of
epigenetics on the development of cancer in humans and
laboratory animals.[5]
So epigenetics is changing our traditional picture of common
chemicals, like DDT. DDT is a powerful environmental toxin --
once it enters a living thing it mimics the behavior of natural
hormones -- resulting in abnormal sexual and reproductive
development. Widespread use of DDT in the 1940s and 1950s is
associated with large scale declines in some bird populations
(like the Peregrine falcon) because DDT causes birds' eggshells
to thin, and thus the eggs crack before the embryo can develop
into a chick.
When persistent environmental pollutants (like DDT) are phased
out, we might be falsely lulled into believing that we have
solved the problem. The thinking is logical -- remove the toxin
from the environment and you get rid of the toxic effects. Not
so according to the findings of Skinner and his colleagues.
The Skinner study tells us that phasing out dangerous toxins
doesn't end the problem -- because the damage done by exposures
decades ago could still flow from generation to generation via
epigenetic pathways.
Skinner and his colleagues treated groups of pregnant rats,
some with methoxychlor and some with vinclozolin. Methoxychlor
is a replacement for DDT, a pesticide used on crops and
livestock and in anima! l feed. Vinclozolin is a fungicide widely
used in the wine industry. It is just one of a suite of widely
used chemicals from flame-retardants to ingredients in plastics
that can cause reproductive abnormalities in laboratory
animals.
Both methoxychlor and vinclozolin are known hormone disruptors
(see Rachel's 486, 487, 499, 501, and 547). Male offspring of
these pesticide-treated mothers had reduced fertility (lower
sperm count, reduced sperm quality), which was not a surprising
finding. The scientists then bred these offspring, and again
the male offspring had reduced fertility. This came as a complete
surprise. Over 90% of the male offspring in four generations of
the test animals had reduced fertility.
Skinner's report concludes that genetic mutations are highly
unlikely to produce such a strong signal in the treated animals
and that DNA-methylation is the likely mechanism responsible
for the observed decline in male fertility.
Treating the mother rats during pregnancy apparently
re-programmed the genetic material in the male offspring so
that all subsequent male offspring suffered lower fertility
from this environmental factor.
Skinner believes that his findings in rats could explain the
dramatic rise in breast and prostate cancers in humans in
recent decades (see Rachel's 346, 369, 375, 385 and 547) as
partly due to the cumulative effects of multiple toxins over
several generations.
Skinner acknowledges that the doses he gave his rats were high,
compared to the doses humans might expect to receive from
environmental exposures. He is continuing his rat experiments
with lower doses now.
Of course all this new information makes the control of toxic
chemicals even more important that previously thought. The
health of future generations is at stake.
The development of epigenetics also greatly complicates
toxicity tes! ting, and chemical risk assessment. Epigenetics
tells us that much additional toxicity testing will
be needed. So far, there are no standardized,
government-approved protocols for conducting epigenetic tests.
Until such protocols emerge (which could take years), and a
great deal of expensive testing has been completed (requiring
many more years), risk assessors will have to acknowledge that
-- so far as epigenetics is concerned -- they are flying blind.
=====
* Tim Montague is Associate Director of Environmental Research
Foundation. He holds an M.S. degree in ecology from
University of Wisconsin-Madison and lives in Chicago.
[1] Here we define a genetic mutation as a change in the
sequence of nucleotide bases (C,A,T,G). We recognize that
epigenetic changes are heritable changes to the DNA, but they
are not sequence changes.
[2] To see nine articles on epigenetics from the popular press,
including an excellent series from the Wall Street Journal, go
to [
www.rachel.org]
[3] M. Anway, A. Cupp, M. Uzumcu, and M. Skinner, "Epigenetic
Transgenerational Actions of Endocrine Disruptors and Male
Fertility," SCIENCE Vol. 308 (June 3, 2005), pgs. 1466-1469.
Michael Skinner is director of the University of Washington's
Center for Reproductive Biology; [
www.skinner.wsu.edu]
[4] Andrew Feinberg and Bert Vogelstein, "Hypomethylation
distinguishes genes of some human cancers from their normal
counterparts," NATURE Vol. 301 (January 6, 1983), pgs. 89-92.
[5] Andrew Feinberg and Benjamin Tycko, "The history of cancer
epigenetics," NATURE REVIEWS (February 2004) Vol. 4, pgs.
143-153.
ORGANOCHLORINES:
These pesticides are chlorine containing compounds including DDT, aldrin, dieldrin and lindane. The organochlorines act through disruption of neurotransmission. PCB's, which are not used as pesticides, are also organochlorines with similar human action and thus have the potential for an additive effect.
The greatest concern with the organochlorines are the long term effects. The U.S. EPA has concluded that DDT, DDE and DDD are probable human carcinogens. On this basis both Canada and the U.S. banned the organochlorines; however, they continue to be very prevalent posing long- term health risks.
The organochlorines are still widely used in developing countries including Central and South America, India, China and many other countries. Products imported from these countries are obvious sources of DDT and other organochlorines. They are also transported in air, oceans and bioaccumulate in organisms.15
Food is the primary route of exposure. Foods which may contain DDT include: meat, fish and poultry, dairy products and root and leafy vegetables. Fish from the Great Lakes Basin and inland waters are a large food source of organochlorine exposure.
A study of the concentrations and dietary intake of selected organochlorines in fresh food composites grown in Ontario demonstrated that organochlorine residues were detected in all of the food composites. This included all types of fresh food grown in Ontario including beef, poultry, fruits and vegetables (it did not include fish). The Findings suggest that consumption of eggs and meat is also a significant source of exposure to the majority of organochlorine chemicals studied.16
[
www.caps.20m.com]